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c6 rat glioma cells  (ATCC)


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    Structured Review

    ATCC c6 rat glioma cells
    C6 Rat Glioma Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1959 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/c6+rat+glioma+cells/C6/pmc13092973-33-0-4
    Average 96 stars, based on 1959 article reviews
    c6 rat glioma cells - by Bioz Stars, 2026-10
    96/100 stars

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    Related Articles

    Cell Culture:

    Article Title: rRNA expansion segments mediate ribosome dimerization as a conserved stress response
    Article Snippet: .. C6 rat glioma cells (ATCC, CCL-107) were cultured in high-glucose DMEM (Gibco #11960044) supplemented with 10% FBS, 2 mM L-glutamine, and antibiotics (penicillin and streptomycin, Gibco #15070063). ..

    Article Title: rRNA expansion segments mediate ribosome dimerization as a conserved stress response.
    Article Snippet: .. C6 rat glioma cells (ATCC, CCL-107) were cultured in high-glucose DMEM (Gibco #11960044) supplemented with 10% FBS, 2 mM L-glutamine, and antibiotics (penicillin and streptomycin, Gibco #15070063). ..

    Article Title: rRNA Expansion Segments Mediate Ribosome Dimerization as a Conserved Stress Response
    Article Snippet: .. C6 rat glioma cells (ATCC, CCL-107) were cultured in high-glucose Dulbecco’s Modified Eagle’s Medium (DMEM, Gibco #11960044) supplemented with 10% fetal bovine serum (FBS), 2 mM L-glutamine, and antibiotics (penicillin and streptomycin, Gibco #15070063). ..

    Modification:

    Article Title: Theranostic Angiopep-2-Conjugated FeTaO x @Au Core-Shell Magnetic Nanoparticles for Glioma Treatment and Dual Medical Imaging.
    Article Snippet: .. C6 rat glioma cells (CCL107; ATCC) and L929 mouse fibroblasts (CCL1; ATCC) were grown in Dulbecco’s modified Eagle’s medium (DMEM) supplemented with 10% fetal bovine serum (GIBCO). ..

    Article Title: Theranostic Angiopep-2-Conjugated FeTaO x @Au Core–Shell Magnetic Nanoparticles for Glioma Treatment and Dual Medical Imaging
    Article Snippet: .. C6 rat glioma cells (CCL107; ATCC) and L929 mouse fibroblasts (CCL1; ATCC) were grown in Dulbecco’s modified Eagle’s medium (DMEM) supplemented with 10% fetal bovine serum (GIBCO). ..

    Article Title: rRNA Expansion Segments Mediate Ribosome Dimerization as a Conserved Stress Response
    Article Snippet: .. C6 rat glioma cells (ATCC, CCL-107) were cultured in high-glucose Dulbecco’s Modified Eagle’s Medium (DMEM, Gibco #11960044) supplemented with 10% fetal bovine serum (FBS), 2 mM L-glutamine, and antibiotics (penicillin and streptomycin, Gibco #15070063). ..

    Article Title: Formulation-Dependent Cytotoxic Effects of Commercial and Analytical-Grade Mancozeb in C6 Glial Cells.
    Article Snippet: Commercial MZ (manganese/zinc ethylene bisdithiocarbamate) was obtained as Unizeb Gold (UPL do Brasil, Indústria e Comércio de Insumos Agropecuários S.A.), a water- dispersible granule (WG) formulation containing 750 g/kg (75% w/w) of the active ingredient, and analytical- grade MZ (Pestanal) was purchased from SigmaAldrich, USA. .. C6 rat glioma cells (ATCC CCL- 107) were obtained from the American Type Culture Collection (Rockville, MD, USA) and cultured in flasks containing Dulbecco's Modified Eagle's Medium (DMEM) supplemented with 5% (v/v) fetal bovine serum (FBS) and an antibiotic–antimycotic solution (100x), diluted 1:100 to obtain final concentrations of penicillin (100 U/ mL), streptomycin (100 μg/mL), and amphotericin B (0.25 μg/ mL). ..

    Membrane:

    Article Title: Advancing Brain Health Naturally: β-Caryophyllene and Xanthohumol as Neuroprotective Agents
    Article Snippet: .. To evaluate the protective effects of BCP against glutamate-induced cytotoxicity in C6 glioma cells. , Cells: C6 rat glioma cells (ATCC CCL-107). Treatments: Glu-induced cytotoxicity: Cells exposed to 0.05–10 mM Glu for 24 h. BCP co-administration: Glu (1 mM) + BCP (0.5–1 μM) for 1 or 24 h. BCP pretreatment: BCP (0.5–3 μM) for 24 h before Glu (1 mM) exposure. , BCP inhibited ROS production and reestablished the mitochondrial membrane potential (CB2R/Nrf2 signaling pathway), which led to the prevention of C6 glioma cell line from Glu-induced cytotoxicity. , BCP activated Nrf2 and improved antioxidant defenses, partly via CB2 receptor activation. , [ ] . .. To investigate whether BCP promotes neuritogenesis independently of CB2 receptor activation. , Cell lines: PC12 and SH-SY5Y. Treatment: PC12 cells treated with BCP (10–50 μM) ± NGF or K252a (TrkA inhibitor) for 72 h; SH-SY5Y cells treated with BCP (10 μM) ± retinoic acid for 72 h. , BCP stimulated the process of neuritogenesis and synaptogenesis in PC12 cells through a mechanism that did not involve CB2 receptors. , Activation of NGF-specific receptor trkA. Neuritogenesis and axonal protein upregulation, independent of CB2 or NGF expression. , [ ] .

    Activation Assay:

    Article Title: Advancing Brain Health Naturally: β-Caryophyllene and Xanthohumol as Neuroprotective Agents
    Article Snippet: .. To evaluate the protective effects of BCP against glutamate-induced cytotoxicity in C6 glioma cells. , Cells: C6 rat glioma cells (ATCC CCL-107). Treatments: Glu-induced cytotoxicity: Cells exposed to 0.05–10 mM Glu for 24 h. BCP co-administration: Glu (1 mM) + BCP (0.5–1 μM) for 1 or 24 h. BCP pretreatment: BCP (0.5–3 μM) for 24 h before Glu (1 mM) exposure. , BCP inhibited ROS production and reestablished the mitochondrial membrane potential (CB2R/Nrf2 signaling pathway), which led to the prevention of C6 glioma cell line from Glu-induced cytotoxicity. , BCP activated Nrf2 and improved antioxidant defenses, partly via CB2 receptor activation. , [ ] . .. To investigate whether BCP promotes neuritogenesis independently of CB2 receptor activation. , Cell lines: PC12 and SH-SY5Y. Treatment: PC12 cells treated with BCP (10–50 μM) ± NGF or K252a (TrkA inhibitor) for 72 h; SH-SY5Y cells treated with BCP (10 μM) ± retinoic acid for 72 h. , BCP stimulated the process of neuritogenesis and synaptogenesis in PC12 cells through a mechanism that did not involve CB2 receptors. , Activation of NGF-specific receptor trkA. Neuritogenesis and axonal protein upregulation, independent of CB2 or NGF expression. , [ ] .



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